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A long virus to be harmless, actually can play a role in Parkinson’s disease, The condition affects more than a million Americans.
Scientists of northwest medicine have discovered human pegivirus (HPGV) in the brain and spinal fluid of people with Parkinson’s, but not in those without diseases. The results challenge decades of viruses.
“HPGV is a common symptom infection without a symptom that has been previously known to infect the brain”, the chief of neuroinfective diseases in northwest, said in a statement to the public.
“We were surprised to find him in the Parkinson’s brain on such a high frequency, not in control.”
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Researchers reviewed the post-mortem brain of 10 patients with 10 Parkinson and 14 people who died of other causes. (East)
The findings were published in the JCCs’ journal insight.
Researchers reviewed the post-mortem brain of 10 patients with 10 Parkinson and 14 people who died of other causes. The virus appeared in five of 10 parkinsons of brains and none of 14 controls.
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Also appeared in spinal fluid samples, suggesting that the virus could be active in nervous system. Patients who carry HPGV showed more advanced brain changes related to Parkinson, including protein and changed brain chemistry.
The team did not stop in the brain tissue. Using blood samples from more than 1,000 participants in the project that leads Michael J. Fox Foundations, researchers saw the same immune system changes associated with the virus.
Researchers also examined blood samples of 1,000 people with Parkinson, in an effort to follow the virus. (East)
More striking: Mutation patients in connection with Parkinson’s regarding Gina LRRK2, otherwise reacted to HPGV from those without mutation.
“This suggests that it could be a factor of environmental protection that communicates with the body in the way we have not realized earlier,” said the Watern.
“May affect how Parkinson develops, especially in people with certainty Genetic backgrounds. “
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Parkinson’s disease is the second most common brain disorder after Alzheimer, according to an alleged institute for neurological disorders and stroke. Although a small percentage of cases inherited, most patients have no family history, and the cause remained unknown.
“It is too early to say that the virus causes the disease,” says Dr. Joel Slinas, a neurologist in behavior. (East)
If HPGV really plays a role, it could help explain why some people develop Parkinson, while others do not work. It could also open the door to new treatments that target viruses or immune systems.
“The study was more often revealed by HPGV traces in the brain of people with Parkinson’s disease than in control. This raises the possibility of a relationship between viral exposure and parkinsons, but it is too early virus It causes disease, “dr. Joel Slinas, he was a neurologist in behavior and an associate professor in Niu Grossman, said Fox News Digital.
Sling, who was not involved in the study, also said that much major and long-term studies will need to determine whether this association has a real clinical significance.
“For now, people should know that this is an early research, not a reasonable evidence, but this virus causes Parkinson, similar to how the job on HerpesViruses in Alzheimer, proposed a possible connection, but remains to be fully determined.”
The northwest team plans to expand his studio to see how often HPGV is in people with Parkinson’s. (East)
According to Parkinson’s Foundation, almost 90,000 Americans diagnose every year. That number is expected to reach 1.2 million to 2030. years.
The northwest team plans to expand his study to see how many HPGV is in people with Parkinson’s versus healthy controls, and whether other viruses can be included.
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“For a virus who found it harmless, these findings suggest that they can have important effects in the context of Parkinson’s disease,” said the Secretary.
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“We also aim to understand how viruses and genes communicate; insights that could reveal how Parkinson’s begins and can help guide future therapies.”
